1. Garrett SR, Mietrach N, Deme J, Bitzer A, Yang Y, Ulhuq FR, Kretschmer D, Heilbronner S, Smith TK, Lea SM, Palmer T. A type VII-secreted lipase toxin with reverse domain arrangement. Nat Commun. 2023;14(1):8438. doi: 10.1038/s41467-023-44221-y.
2. Slavetinsky J, Lehmann E, Slavetinsky C, Gritsch L, van Dalen R, Kretschmer D, Bleul L, Wolz C, Weidenmaier C, Peschel A. Wall Teichoic Acid Mediates Staphylococcus aureus Binding to Endothelial Cells via the Scavenger Receptor LOX-1. ACS Infect Dis. 2023;9(11):2133-2140. doi: 10.1021/acsinfecdis.3c00252.
3. Kengmo Tchoupa A, Kretschmer D, Schittek B, Peschel A. The epidermal lipid barrier in microbiome-skin interaction. Trends Microbiol. 2023;31(7):723-734. doi: 10.1016/j.tim.2023.01.009.
4. Lebtig M, Scheurer J, Muenkel M, Becker J, Bastounis E, Peschel A, Kretschmer D. Keratinocytes use FPR2 to detect Staphylococcus aureus and initiate antimicrobial skin defense. Front Immunol. 2023;14:1188555. doi: 10.3389/fimmu.2023.1188555.
5. Kengmo Tchoupa A, Kretschmer D, Schittek B, Peschel A. The epidermal lipid barrier in microbiome-skin interaction.Trends Microbiol. 2023 Feb Review.
6. Kretschmer D, Breitmeyer R, Gekeler C, Lebtig M, Schlatterer K, Nega M, et al. Staphylococcus aureus Depends on Eap Proteins for Preventing Degradation of Its Phenol-Soluble Modulin Toxins by Neutrophil Serine Proteases. Front Immunol. 2021;12:701093. doi: 10.3389/fimmu.2021.701093.
7. Schlatterer K, Beck C, Schoppmeier U, Peschel A, Kretschmer D. Acetate sensing by GPR43 alarms neutrophils and protects from severe sepsis. Commun Biol. 2021;4(1):928. doi: 10.1038/s42003-021-02427-0.
8. Weiss E, Schlatterer K, Beck C, Peschel A, Kretschmer D. Formyl-peptide receptor activation enhances phagocytosis of community-acquired methicillin-resistant Staphylococcus aureus. J Infect Dis. 2019. doi: 10.1093/infdis/jiz498.
9. Schlatterer K, Beck C, Hanzelmann D, Lebtig M, Fehrenbacher B, Schaller M, Ebner P, Nega M, Otto M, Kretschmer D, Peschel A. The Mechanism behind Bacterial Lipoprotein Release: Phenol-Soluble Modulins Mediate Toll-Like Receptor 2 Activation via Extracellular Vesicle Release from Staphylococcus aureus. mBio. 2018;9(6). doi: 10.1128/mBio.01851-18.
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